GLP-(Cag) + GLP-3 Blend

Description

Cagrilintide + Retatrutide 5mg/5mg Blend (Amylin / GLP-1 / GIP / Glucagon Receptor Co-Agonist System)

Concentration: 5 mg Cagrilintide + 5 mg Retatrutide per vial
Form: Lyophilized Powder
Storage: Store at −20 °C. Protect from light and moisture.

Molecular Formula (Cagrilintide): C₁₈₇H₂₈₉N₅₁O₅₉
Molecular Weight (Cagrilintide): 4,046.67 g/mol
CAS Number (Cagrilintide): 1339617-40-4

Molecular Formula (Retatrutide): C₁₈₇H₂₈₆N₄₆O₅₉
Molecular Weight (Retatrutide): 4,766.54 g/mol
CAS Number (Retatrutide): — (Pending / Not Assigned)

Purity: ≥98 % (HPLC)

Description

The Cagrilintide + Retatrutide 5/5 Blend is a next-generation poly-hormonal peptide system combining amylin receptor agonism with triple incretin and glucagon receptor activation, providing a powerful research platform for studying energy homeostasis, adiposity modulation, and metabolic regulation.

• Cagrilintide is a long-acting amylin analog that suppresses appetite, delays gastric emptying, and enhances satiety through calcitonin receptor and amylin receptor signaling.

• Retatrutide is a GLP-1 / GIP / Glucagon tri-agonist that integrates incretin and glucagon pathways, driving insulin sensitivity, lipid oxidation, and increased thermogenesis.

In combination, these peptides deliver multi-receptor synergy, amplifying the effects of incretin signaling while engaging energy-expending glucagon pathways and appetite-reducing amylin circuits.
This blend provides an advanced tool for research into body-weight regulation, glucose control, and metabolic rate enhancement in preclinical systems.

Intended Use

This product is intended for laboratory research use only.
It is not for human consumption, diagnostic, or therapeutic applications.
All handling should be performed by qualified professionals in a controlled research environment.

Key Research Applications

• Investigation of multi-receptor co-agonism (GLP-1, GIP, Glucagon, Amylin) in metabolic control

• Studies on energy expenditure, thermogenesis, and lipid oxidation mechanisms

• Research on appetite suppression, glycemic stability, and body-composition change

• Analysis of integrated neuroendocrine pathways in metabolic homeostasis