Selank + Semax Blend

Description

Selank + Semax 10mg/10mg Blend (Nootropic & Neuroregenerative Neuropeptide Complex)

Concentration: 10 mg Selank + 10 mg Semax per vial
Form: Lyophilized Powder
Storage: Store at −20 °C. Protect from light and moisture.

Molecular Formula (Selank): C₃₃H₅₇N₁₁O₉
Molecular Weight: 751.9 g/mol
CAS Number (Selank): 129954-34-3

Molecular Formula (Semax): C₃₇H₅₁N₉O₁₀S
Molecular Weight: 869.0 g/mol
CAS Number (Semax): 80714-61-0

Purity: ≥98% (HPLC)

Description

The Selank + Semax 10/10 Blend is a synergistic neuropeptide combination designed for advanced research into neuroprotection, cognitive enhancement, and stress adaptation.
These two heptapeptides complement one another by targeting distinct yet converging neurotrophic and neurotransmitter-modulating pathways in the central nervous system.

• Selank (a synthetic analog of tuftsin) exerts anxiolytic and neuromodulatory effects through regulation of GABA, serotonin, and dopamine transmission, while also modulating immune–neural signaling and BDNF expression.

• Semax (an ACTH(4–10) analog with a Pro-Gly-Pro modification) enhances neurotrophin synthesis (BDNF, NGF), supports synaptic plasticity, and exhibits cognitive-enhancing and neuroprotective properties via modulation of dopaminergic and glutamatergic pathways.

When combined, these peptides demonstrate synergistic action on cognitive function, mood regulation, oxidative resilience, and neuronal recovery, making this blend an ideal model for studying integrated neuroadaptive and nootropic mechanisms.

Intended Use

This product is intended for laboratory research use only.
It is not for human consumption, diagnostic, or therapeutic applications.
All handling should be performed by qualified professionals in a controlled research environment.

Key Research Applications

• Investigation of neurotrophic peptide synergy and synaptic plasticity

• Studies on stress adaptation, anxiety modulation, and neurotransmitter balance

• Research on BDNF/NGF regulation and neuronal repair mechanisms

• Analysis of combined nootropic and neuroprotective signaling pathways